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Pathogenesis of fetal and neonatal alloimmune thrombocytopenia and mechanisms of IVIG and anti-FcRn therapies

Pathogenesis of fetal and neonatal alloimmune thrombocytopenia and mechanisms of IVIG and anti-FcRn therapies
胎儿和新生儿同种免疫性血小板减少症的发病机制以及 IVIG 和抗 FcRn 治疗的机制
批准号：
239359
负责人：
Ni Heyu
金额：
$40.6万
依托单位：
Unity Health Toronto
依托单位国家：
加拿大
项目类别：
Operating Grants
财政年份：
2011
资助国家：
加拿大
项目状态：
已结题
起止时间：
2011-09-01 至 2015-09-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=239359&lang=en
关键词：
Angiogenesis And Fetal Bleeding Disorders Antibodies And Cellular Immune Responses Fetal And Neonatal Alloimmune Thrombocytopenia Intravenous Immunoglobulin G (Ivig) And Anti-Fcrn Antibody Novel Animal Models Platelet Destruction And Phagocytosis

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中文摘要
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英文摘要
Platelets are small blood cells that are responsible for the arrest of bleeding at the site of vessel injury. A low platelet count is called thrombocytopenia. Antibodies to fetal platelets can be induced in the mother by proteins called antigens on platel
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