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Embryonic DNA repair and ethanol-initiated behavioural deficits in oxoguanine glycosylase 1 (OGG1) knockout mice: A role of oxidatively damaged DNA and protection by a free radical spin trapping agent

Embryonic DNA repair and ethanol-initiated behavioural deficits in oxoguanine glycosylase 1 (OGG1) knockout mice: A role of oxidatively damaged DNA and protection by a free radical spin trapping agent
氧代鸟嘌呤糖基化酶 1 (OGG1) 敲除小鼠的胚胎 DNA 修复和乙醇引发的行为缺陷：氧化损伤 DNA 的作用和自由基自旋捕获剂的保护
批准号：
269110
负责人：
Miller Lutfiya
金额：
$0.07万
依托单位：
University of Toronto
依托单位国家：
加拿大
项目类别：
财政年份：
2012
资助国家：
加拿大
项目状态：
已结题
起止时间：
2012-09-01 至 2012-12-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=269110&lang=en
关键词：
Alcohol Behavioural Deficits Dna Repair Fetal Alcohol Spectrum Disorder Oxidative Stress Oxidatively Damaged Dna

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While it is widely known that alcohol consumption during pregnancy is harmful to the unborn child, the nature of the damage and how it occurs is not known, precluding the identification of high-risk children and potential therapeutic strategies for preven
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