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Ketamine-induced NMDA receptor hypofunction in nonhuman primates as a model for cognitive deficits in schizophrenia

Ketamine-induced NMDA receptor hypofunction in nonhuman primates as a model for cognitive deficits in schizophrenia
氯胺酮诱导的非人灵长类 NMDA 受体功能减退作为精神分裂症认知缺陷的模型
批准号：
339376
负责人：
Ma Liya
金额：
$9.83万
依托单位：
University of Western Ontario
依托单位国家：
加拿大
项目类别：
Fellowship Programs
财政年份：
2015
资助国家：
加拿大
项目状态：
已结题
起止时间：
2015-11-01 至 2018-11-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=339376&lang=en
关键词：
Animal Model Computational Property Electrophysiology Pharmacology Prefrontal Cortex Schizophrenia Working Memory

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英文摘要
Most patients suffering from schizophrenia have cognitive problems. One especially debilitating problem, the working memory deficit, is the impairment in their ability to hold information temporarily on-line for planning, making decisions and interacting
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Canada Research Chair - Tier 2