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Knockdown of the Mixed Lineage Kinase Domain-Like Protein (MLKL) Enhances Extramedullary Hematopoiesis to Increase Atherosclerosis

Knockdown of the Mixed Lineage Kinase Domain-Like Protein (MLKL) Enhances Extramedullary Hematopoiesis to Increase Atherosclerosis
混合谱系激酶结构域样蛋白 (MLK​​L) 的敲低可增强髓外造血,从而加剧动脉粥样硬化
批准号:
400350
负责人:
Rasheed Adil
金额:
$0.07万
依托单位国家:
加拿大
项目类别:
财政年份:
2019
资助国家:
加拿大
项目状态:
已结题
起止时间:
2019-01-01 至 2020-01-01

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中文摘要
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英文摘要
Atherosclerosis is the greatest contributor to death and is characterized by increased blood cholesterol, as well as inflammation in the heart, and is typically recognized as a narrowing of vessels in the heart by a build up of cholesterol-rich plaques. D
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Hyperlipidemia-induced Hematopoiesis is Repressed by MLKL in Endothelial Cells of the Splenic Niche
Uncovering novel regulators of splenic hematopoiesis during cardiovascular disease
  • 批准号:
    473770
  • 项目类别:
    Fellowship Programs
  • 资助金额:
    $3.28万
  • 财政年份:
    2022
  • 负责人:
    Rasheed Adil
  • 依托单位:
海外基金