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Matricellular protein CCN5/WISP2 mediates a synergistic stimulation of pancreatic beta-cell expansion by IGF-I and Wnt signaling

Matricellular protein CCN5/WISP2 mediates a synergistic stimulation of pancreatic beta-cell expansion by IGF-I and Wnt signaling
基质细胞蛋白 CCN5/WISP2 通过 IGF-I 和 Wnt 信号传导协同刺激胰腺 β 细胞扩增
批准号：
431555
负责人：
Liu Jun-Li
金额：
$7.29万
依托单位：
Research Institute of the McGill University Health Centre
依托单位国家：
加拿大
项目类别：
Operating Grants
财政年份：
2020
资助国家：
加拿大
项目状态：
已结题
起止时间：
2020-03-01 至 2021-03-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=431555&lang=en
关键词：
Beta Cell Regeneration Cell Survival Celll Surface Receptor Gene-Targeting Human Islets Insulin Secretion Molecular Mechanism Pancreatic Islets Proliferation Transgenic Mice

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英文摘要
Pancreatic beta cells secrete insulin which controls energy metabolism; lack of beta cells or insulin secretion causes diabetes mellitus. Studies on the regeneration of pancreatic islets, using animal models, represent a major effort leading to a cure of
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Matricellular protein CCN5/WISP2 mediates a synergistic stimulation of pancreatic beta-cell expansion by IGF-I and Wnt signaling
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