菊花是全球重要的花卉作物，开花受短日照条件诱导。菊花的周年供应主要依赖于人工的光周期调节，不仅费时、费力而且能耗高。研究菊花的成花机理，控制开花品质，创制光周期不敏感菊花新种质一直是本领域研究的热点。申请者前期研究发现，光周期途径中的关键信号蛋白BBX24 (CO家族)可通过转调赤霉素（GA）途径实现菊花在非开花诱导长日照条件下的成花转变；而GA受体基因GID1s作为BBX24的下游组分，参与了对菊花提早开花的调节。本研究拟进一步解析GID1s信号通道调节菊花成花的分子调控模式，从转录和转录后层面，系统探索GID1s参与光周期途径的信号传导进而影响菊花成花的分子机制。本研究可为节能型精准调控花期的周年生产提供理论依据，为培育光周期不敏感的菊花新品种提供基因储备，也为短日照植物菊花的开花生物学研究提供新数据。

Chrysanthemum is an important ornamental crop worldwide, is a plant in which short-day hastens time to flowering. The year-round production of chrysanthemum depends mainly on artificial photoperiod regulation, which is not only time-consuming but also power-wasting. Therefore, studying on the mechanisms of flowering, controling flower quality, as well as creating photoperiod-insensitive germplasms are important aspects in the research filed of chrysanthemum. Our previous work demonstrated that CO family member BBX24 in chrysanthemum modulates flowering transition under non-inductive long-day condition through gibberellin pathway. In addition, gibberellin receptors GID1s as downstream genes of BBX24, participate in promoting flowering of chrysanthemum. Here, we aim to elucidate the regulatory mechanism of GID1s participating in photoperiod pathway to influence the flowering time of chrysanthemum on the transcriptional and post-transcriptional levels. Our results will be valuable for understanding the molecular mechanism of flowering in the typical obligate short-day plant chrysanthemum, and will provide the candidate genes for breeding photoperiod-insensitive chrysanthemum cultivars. Meanwhile，it also provides new data for molecular biology analysis of chrysanthemum blossom.