Cognitive specificity in internalizing and externalizing problems in community and clinic-referred children
Cognitive specificity in internalizing and externalizing problems in community and clinic-referred children
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DOI：
10.1207/s15374424jccp2902_6
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发表时间：
2000-06-01
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影响因子：
--
通讯作者：
Epkins, CC
中科院分区：
文献类型：
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作者：
Epkins, CC
检查了 Beck (1967) 抑郁症模型中的认知特征及其认知内容特异性假说 (Beck, Brown, Steer, Eidelson, & Riskind, 1987) 是否是区分内在问题和外在问题的宽带特定特征。从社区（n = 389；196 名男孩，193 名女孩；年龄 M = 9.98，SD = 1.52）和诊所（n = 82；55 名男孩，27 名女孩；年龄 M = 10.42，SD = 2.91）样本中，定义了 4 组行为测量，然后检查认知测量：仅外化（n = 33），仅内化（n = 41），共病外化和内化（n = 53），以及控制（既不外化也不内化；n = 35）。在社区和临床样本中，仅内化组和共病组都报告了比仅外化组和对照组显着更多的认知障碍（消极认知三联体、认知处理扭曲以及“抑郁”和“焦虑”思想内容）。结果与精神病理学的总体水平（如母亲报告的）或儿童报告中的社会期望无关。这些发现表明，贝克模型中的认知特征区分了内化问题和外化问题，并增加了越来越多的文献，这些文献呼吁进一步完善认知模型的概念，以理解窄带和宽带综合症。
Examined whether cognitive features in Beck's (1967) model of depression and his cognitive content-specificity hypothesis (Beck, Brown, Steer, Eidelson, & Riskind, 1987) are broadband specific features that distinguish internalizing problems from om externalizing problems. From a community (n = 389; 196 boys, 193 girls; age M = 9.98, SD = 1.52) and clinic (n = 82; 55 boys, 27 girls; age M = 10.42, SD = 2.91) sample, 4 groups were defined on behavioral measures and then examined on cognitive measures: externalizing only (n = 33), internalizing only (n = 41), comorbid externalizing and internalizing (n = 53), and control (neither externalizing nor internalizing; n = 35). Both the internalizing only and comorbid groups reported significantly more cognitive disturbances (negative cognitive triad, cognitive processing distortions, and "depressive" and "anxious" thought content) than both of the externalizing only and control groups in both the community and clinic samples. The results were not related to either overall level of psychopathology (as reported by mothers) or social desirability in children's reporting. These findings suggest that the cognitive features in Beck's model distinguish internalizing from externalizing problems and add to a growing literature that calls for further conceptual refinement of cognitive models for understanding both narrow-band and broadband syndromes.