Frequency distributions of periodontal attachment loss. Computer simulation.

Frequency distributions of periodontal attachment loss. Computer simulation.
复制标题

牙周附着丧失的频率分布。

DOI:
10.1111/j.1600-051x.1986.tb00855.x
复制
发表时间:
1986
影响因子:
6.7
通讯作者:
Haffajee,AD
Haffajee,AD
中科院分区:
医学1区
文献类型:
--
作者:
Socransky,SS;Haffajee,AD

文献摘要

参考文献

被引文献

相似文献

根据 61 名患有破坏性牙周病受试者的基线附着水平测量频率分布曲线拟合,描述了 3 种附着丧失模式。 30名第一组个体的依恋水平测量的频率分布通过2条正态曲线之和令人满意地拟合30名第一组个体的依恋水平测量结果,而14名第二组个体的依恋水平测量结果通过3条正态曲线之和拟合。 17 名第 III 组受试者的频率分布由单一正态曲线拟合。使用计算机算法通过改变活动病变处的“爆发大小”以及在不同时间段内对受到攻击的部位施加“免疫”来模拟依恋水平损失频率分布。可以使用 0.3 至 3.4 毫米之间的爆发大小来模拟第一组受试者的频率分布。这些受试者可分为 2 组:14 名具有局部最小附着损失的受试者需要 < 1 毫米的平均突发尺寸,而 16 名在患病部位具有更多破坏的受试者需要 > 1 毫米的平均突发尺寸来模拟其频率分布。 17 名第 III 组受试者中的 16 名受试者的频率分布可以使用 < 1 毫米的平均突发尺寸进行模拟。然而,也可以使用更大的突发大小来模拟频率分布,并在受攻击地点长时间施加“免疫”。第二组受试者的频率分布更难以模拟。使用较大的平均突发尺寸 (> 1 mm) 可以获得比使用较小的突发尺寸更好的模拟。然而,最令人满意的拟合是通过将大突发攻击模式叠加在使用小突发创建的分布上来获得的。小突发和大突发的最佳平均大小因受试者而异,突发大小改变的平均附着水平也不同。模拟表明,不同的频率分布是由不同的依恋丧失进展模式引起的,其中突发大小和可能的局部“免疫”发挥了主要作用。
3 patterns of attachment loss were described based on curve fitting the frequency distributions of baseline attachment level measurements of 61 subjects with destructive periodontal diseases. The frequency distributions of attachment level measurements of 30 group I individuals were satisfactorily fit by tachment level measurements of 30 group I individuals were saticfactorily fit by the sum of 2 normal curves, while those of 14 group II individuals were fit by the sum of 3 normal curves. Frequency distributions of 17 group III subjects were fit by a single normal curve. A computer algorithm was used to simulate attachment level loss frequency distributions by varying the “burst size” at active lesions and by imposing “immunity” for varying periods of time on sites which had been attacked.Frequency distributions of group I subjects could be simulated using burst sizes between 0.3 and 3.4 mm. These subjects could be divided into 2 groups: 14 subjects with localized minimal attachment loss required an average burst size of < 1 mm, while 16 subjects with more destruction at diseased sites required an average burst size of > 1 mm to simulate their frequency distributions. Frequency distributions of 16 of 17 group III subjects could be simulated using an average burst size of < 1 mm. However, the frequency distributions could also be simulated using larger burst sizes with the imposition of “immunity” for substantial periods of time at the attacked sites.The frequency distributions of group II subjects were more difficult to simulate. A better simulation could be obtained using a large average burst size (> 1 mm) than a small burst size. However, the most satisfactory fit was obtained by the superimposition of a large burst pattern of attack on a distribution created using small bursts. The optimal average sizes of the small and large bursts varied from subject to subject as did the mean attachment level at which the burst size was changed. The simulations suggest that the different frequency distributions result from different modes of progression of attachment loss with burst size and possibly local “immunity” playing major rôles.
DOI: 10.1111/j.1600-051x.1986.tb00856.x
发表时间: 1986
影响因子: 6.7
作者:
Haffajee,AD;Socransky,SS
通讯作者: Socransky,SS