Gene expression signatures affected by alcohol-induced DNA methylomic deregulation in human embryonic stem cells.
Gene expression signatures affected by alcohol-induced DNA methylomic deregulation in human embryonic stem cells.
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DOI:
10.1016/j.scr.2014.03.009
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发表时间:
2014-05
影响因子:
1.2
通讯作者:
Kim, Yong
中科院分区:
文献类型:
--
作者:
Khalid, Omar;Kim, Jeffrey J.;Kim, Hyun-Sung;Hoang, Michael;Tu, Thanh G.;Elie, Omid;Lee, Connie;Vu, Catherine;Horvath, Steve;Spigelman, Igor;Kim, Yong
Stem cells, especially human embryonic stem cells (hESCs), are useful models to study molecular mechanisms of human disorders that originate during gestation. Alcohol (ethanol, EtOH) consumption during pregnancy causes a variety of prenatal and postnatal disorders collectively referred to as fetal alcohol spectrum disorders (FASDs). To better understand the molecular events leading to FASDs, we performed a genome-wide analysis of EtOH's effects on the maintenance and differentiation of hESCs in culture. Gene Co-expression Network Analysis showed significant alterations in gene profiles of EtOH-treated differentiated or undifferentiated hESCs, particularly those associated with molecular pathways for metabolic processes, oxidative stress, and neuronal properties of stem cells. A genome-wide DNA methylome analysis revealed widespread EtOH-induced alterations with significant hypermethylation of many regions of chromosomes. Undifferentiated hESCs were more vulnerable to EtOH's effect than their differentiated counterparts, with methylation on the promoter regions of chromosomes 2, 16 and 18 in undifferentiated hESCs most affected by EtOH exposure. Combined transcriptomic and DNA methylomic analysis produced a list of differentiation-related genes dysregulated by EtOH-induced DNA methylation changes, which likely play a role in EtOH-induced decreases in hESC pluripotency. DNA sequence motif analysis of genes epigenetically altered by EtOH identified major motifs representing potential binding sites for transcription factors. These findings should help in deciphering the precise mechanisms of alcohol-induced teratogenesis.
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DOI:
10.1111/j.1530-0277.1998.tb05913.x
发表时间:
1998-12-01
期刊:
ALCOHOL-CLINICAL AND EXPERIMENTAL RESEARCH
影响因子:
--
作者:
Foroud, T;Bucholz, KK;Begleiter, H
通讯作者:
Begleiter, H
影响因子:
2.8
作者:
Anthony, Bruce;Zhou, Feng C.;Ruiz, Joseph
通讯作者:
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DOI:
10.1016/j.bbrc.2008.03.060
发表时间:
2008-05-23
影响因子:
3.1
作者:
Fujita, Yuko;Hiroyama, Masami;Tanoue, Akito
通讯作者:
Tanoue, Akito
DOI:
10.1111/j.1530-0277.2002.tb02544.x
发表时间:
2002-03-01
影响因子:
3.2
作者:
Bielawski, DM;Zaher, FM;Abel, EL
通讯作者:
Abel, EL
DOI:
10.1111/j.1530-0277.1991.tb00536.x
发表时间:
1991-06-01
影响因子:
3.2
作者:
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通讯作者:
LIEBER, CS