Inhibition of BAFF binding to its receptor, BR3, with low molecular weight compounds results in suppression of activation of monocytes and B cells in vitro and in vivo model of autoimmune diseases.
Inhibition of BAFF binding to its receptor, BR3, with low molecular weight compounds results in suppression of activation of monocytes and B cells in vitro and in vivo model of autoimmune diseases.
复制标题
用低分子量化合物抑制 BAFF 与其受体 BR3 的结合，可抑制自身免疫性疾病体外和体内模型中单核细胞和 B 细胞的活化。
DOI：
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发表时间：
2017
期刊：
影响因子：
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通讯作者：
Tsutomu Takeuchi
中科院分区：
文献类型：
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作者：
Keiko Yoshimoto;Katsuya Suzuki;Kunio Sugahara;Tsutomu Takeuchi
