Hand-mouth coordination, congenital absence of limb, and evidence for innate body schemas
Hand-mouth coordination, congenital absence of limb, and evidence for innate body schemas
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DOI：
10.1006/brcg.1998.1020
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发表时间：
1998-10-01
影响因子：
2.5
通讯作者：
Cole, J
中科院分区：
文献类型：
--
作者：
Gallagher, S;Butterworth, GE;Cole, J
对先天性（再生障碍性）肢体缺失病例中幻肢的研究提供的关于造成幻肢的先天神经学基质的证据不足。在这项研究中，我们回顾了子宫内和婴儿早期手-口协调的超声和行为研究，灵长类动物的神经生物学研究以及截肢后神经重组的研究。我们提出两个互补的假说来解释aplasic幻影。首先，再生障碍性幻肢是基于与先天运动图式相关的特定神经回路的存在，例如负责早期手-口协调的神经基质。第二，aplasic phantomy修改的机制，涉及到一个复杂的网络，涉及皮层和皮层下结构的神经表征的失踪肢体的重组。(C)北京：科学出版社.
Studies of phantom limb in cases of congenital (aplasic) absence of limb have provided inadequate evidence concerning the innate neurological substrate responsible for the phantom. In this study we review evidence from ultrasonic and behavioral studies of hand-mouth coordination in utero and in early infancy, neurobiological studies in primates, and studies of neural reorganization following amputation. We suggest two complementary hypotheses to explain aplasic phantoms. First, aplasic phantoms are based on the existence of specific neural circuitry associated with innate motor schemas, such as the neural matrix responsible for early hand-mouth coordination. Second, aplasic phantoms are modified by mechanisms that involve a reorganization of neural representations of the missing limb within a complex network involving both cortical and subcortical structures. (C) 1998 Academic Press.