INCORPORATION OF MALONYL COA INTO FATTY ACIDS BY LIVER IN STARVATION AND ALLOXAN-DIABETES

INCORPORATION OF MALONYL COA INTO FATTY ACIDS BY LIVER IN STARVATION AND ALLOXAN-DIABETES
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DOI:
10.1016/0006-291x(60)90169-8
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发表时间:
1960-01-01
影响因子:
3.1
通讯作者:
HUBBARD, DD
HUBBARD, DD
中科院分区:
生物学4区
文献类型:
--
作者:
GIBSON, DM;HUBBARD, DD

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糖尿病和饥饿与肝脏将丙二酰辅酶A直接转化为长链脂肪酸的显著酶缺陷有关。通过用胰岛素预处理糖尿病动物或通过再喂养饥饿的动物来恢复酶损伤。
Diabetes and starvation are associated with a marked enzymic defect in the direct conversion of malonyl CoA to long chain fatty acids by liver. The enzyme impairment is restored by pretreating the diabetic animal with insulin or by refeeding the starved animal.