Fructose 2,6-bisphosphate in livers of genetically obese rats.
Fructose 2,6-bisphosphate in livers of genetically obese rats.
复制标题
遗传性肥胖大鼠肝脏中的果糖 2,6-二磷酸。
作者：
L. Hue;G. van de Werve;B. Jeanrenaud
遗传性肥胖（FA/FA）大鼠的肝脏，饥饿24小时，含有更多的果糖2，6-二磷酸，葡萄糖6-磷酸，果糖6-磷酸和糖原，和更多的丙酮酸激酶和磷酸果糖激酶2的活动，比对照瘦大鼠的肝脏。这些变化可以解释在环磷酸腺苷浓度，这是减少肥胖饥饿大鼠的肝脏。
Livers of genetically obese (fa/fa) rats, starved for 24 h, contained more fructose 2,6-bisphosphate, glucose 6-phosphate, fructose 6-phosphate and glycogen, and more pyruvate kinase and phosphofructokinase 2 activities, than livers of control lean rats. These changes may be explained in terms of cyclic AMP concentration, which was decreased in livers of obese starved rats.