Vascular Biology, Atherosclerosis and Endothelium Biology Myeloid Cells in Infantile Hemangioma
Vascular Biology, Atherosclerosis and Endothelium Biology Myeloid Cells in Infantile Hemangioma
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控制快速自发退化的确切机制遵循在报告中，我们证明了在增殖性血管瘤中存在大量的髓系造血细胞，并提出了一种观察到的由低氧触发的涉及髓系细胞参与的病变演变的机制。我们报道了使用髓系标记物(CD83、CD32、CD14、CD15)意外地共同标记血管瘤内皮细胞的实验结果，提供了这些细胞与正常内皮细胞不同的新证据。(AM Pathol)
The precise mechanisms controlling rapid spontaneous involution follows the of In report we demonstrate the presence of large numbers of hematopoietic cells of the myeloid lineage in proliferating hemangiomas and propose a mechanism for the observed evolution of these lesions that is triggered by hypoxia and involves the participation of myeloid cells. We report the results of experiments using myeloid markers (CD83, CD32, CD14, CD15) that unexpectedly co-labeled hemangioma endothelial cells, providing new evidence that these cells are distinct from normal endothelium. (Am Pathol