Impact of fathers on risky sexual behavior in daughters: A genetically and environmentally controlled sibling study

Impact of fathers on risky sexual behavior in daughters: A genetically and environmentally controlled sibling study
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DOI:
10.1017/s095457941100085x
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发表时间:
2012-02-01
影响因子:
3.3
通讯作者:
Butler, Emily A.
Butler, Emily A.
中科院分区:
心理学2区
文献类型:
--
作者:
Ellis, Bruce J.;Schlomer, Gabriel L.;Butler, Emily A.

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接受较低质量的父亲投资的女孩往往比同龄人从事更危险的性行为(RSB)。虽然父亲投资理论认为这种影响是因果关系,但它可能是由环境或遗传混乱引起的。为了区分这些相互竞争的解释,目前的作者采用了遗传和环境控制的兄弟姐妹设计(N = 101对姐妹;年龄18-36岁),回顾性研究了不同兄弟姐妹暴露于家庭破裂/父亲缺席和父亲质量的影响。与因果关系的解释一致,在生物破坏的家庭中,姐姐和姐妹篇之间的差异是最大的,当姐妹篇之间有一个很大的年龄差距时(从而最大限度地提高对父亲的差异暴露),在家庭中更高质量的父亲作为对RSB的保护因素。此外,父亲质量的低端附近的变化似乎对RSB的影响最大。与此相反,差异兄弟姐妹暴露于家庭破裂/父亲缺席(无论父亲的质量)与RSB无关。差异同胞暴露设计为评价父亲在家庭中的因果效应提供了一种新的准实验方法。
Girls receiving lower quality paternal investment tend to engage in more risky sexual behavior (RSB) than peers. Whereas paternal investment theory posits that this effect is causal, it could arise from environmental or genetic confounds. To distinguish between these competing explanations, the current authors employed a genetically and environmentally controlled sibling design (N = 101 sister pairs; ages 18-36), which retrospectively examined the effects of differential sibling exposure to family disruption/father absence and quality of fathering. Consistent with a causal explanation, differences between older and younger sisters in the effects of quality of fathering on RSB were greatest in biologically disrupted families when there was a large age gap between the sisters (thus maximizing differential exposure to fathers), with greater exposure within families to higher quality fathering serving as a protective factor against RSB. Further, variation around the lower end of fathering quality appeared to have the most influence on RSB. In contrast, differential sibling exposure to family disruption/father absence (irrespective of quality of fathering) was not associated with RSB. The differential sibling-exposure design affords a new quasi-experimental method for evaluating the causal effects of fathers within families.