An investigation of cell wall micropore blocking as a possible mechanism for the decay resistance of anhydride modified wood
An investigation of cell wall micropore blocking as a possible mechanism for the decay resistance of anhydride modified wood
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DOI：
10.1016/j.ibiod.2004.07.003
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发表时间：
2005-01-01
影响因子：
4.8
通讯作者：
Williams, GR
中科院分区：
文献类型：
--
作者：
Hill, CAS;Forster, SC;Williams, GR
科西嘉松（Pinus nigra）边材用乙酸或己酸酐进行化学改性，以增加各种重量百分比。使用溶质排除技术测定化学改性前后木材的细胞壁微孔率。结果表明，随着取代水平的增加，细胞壁微孔率降低，但在高取代水平下，细胞壁仍保持可通性。根据溶质排除数据计算的纤维饱和点 (FSP) 值范围为 c. 40%（对于未改性木材）至 c.大约 20% 25% 的重量百分比增加，但在某种程度上取决于计算方法。有证据表明 FSP 的减少可能是由于键合酰基加合物使细胞壁膨胀所致。结论是，细胞壁中的羟基取代水平并不是酸酐改性木材提供腐烂保护的主要机制。 (C) 2004 Elsevier Ltd. 保留所有权利。
Corsican pine (Pinus nigra) sapwood was chemically modified with acetic, or hexanoic anhydride to a variety of weight percentage gains. The cell wall microporosity of the wood before and after chemical modification was determined using the technique of solute exclusion. The results showed that the cell wall microporosity decreased as the level of substitution increased, but the cell wall remained accessible at high levels of substitution. Values of the fibre saturation point (FSP) calculated from solute exclusion data ranged from c. 40% (for unmodified wood) to c. 20% at approx. 25% weight percentage gain, but were dependent to some degree upon the calculation method. Evidence is presented suggesting that the reduction in FSP may be attributable to bulking of the cell wall by bonded acyl adduct. It is concluded that the level of hydroxyl substitution in the cell wall is not the primary mechanism for giving decay protection in anhydride-modified wood. (C) 2004 Elsevier Ltd. All rights reserved.