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GRK 438: Vascular Biology in Medicine

GRK 438: Vascular Biology in Medicine
GRK 438:医学中的血管生物学
批准号:
272399
负责人:
金额:
$0.0万
依托单位国家:
德国
项目类别:
Research Training Groups
财政年份:
1998
资助国家:
德国
项目状态:
已结题
起止时间:
1997-12-31 至 2006-12-31

项目摘要

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中文摘要
翻译
尽管在预防和治疗方面取得了进展,心血管疾病仍然是世界范围内导致死亡和残疾的最大原因。冠心病、心肌梗死、脑缺血、中风、高血压、移植排斥反应和血管性肾炎是血管壁内炎症、变性和增殖过程的表现。此外,血管突起在肿瘤的形成和转移中起着核心作用。对致病机制的基本了解对于开发诊断、预防和治疗这些疾病的新策略至关重要。“医学中的血管生物学”课程由德国医学联合会资助,慕尼黑大学和工业大学的医学院开发,汇集了高素质的医学预科和研究生,以及生物/生物化学、化学和药学的研究生。毕业生进行血管生物学方面的研究,以获得博士学位(德国医学博士或博士学位)。基于跨学科的方法,该项目为学生提供了获得广泛的科学知识的机会,在生物化学,分子生物学,生理学和细胞生物学的血管壁和血管疾病的发病机制,并有可能接受各种方法的血管细胞生物学的培训,从而接受广泛的教育,在这个中心的生物医学研究领域。
英文摘要
Despite advances in prevention and treatment, cardiovascular disease remains the largest cause of death and disability world-wide. Coronary heart disease, myocardial infarction, cerebral ischemia, stroke, hypertension, transplant rejections and vascular nephritis are manifestations of inflammatory, degenerative and proliferative processes within the vessel wall. Furthermore vascular processes play a central role in tumor formation and metastasis. A fundamental understanding of pathogenic mechanisms is essential for the development of new strategies for diagnosis, prevention, and therapy of these diseases. The program "Vascular Biology in Medicine" financed by the DFG and developed by the medical faculties of the University and Technical University of Munich assembles highly qualified pre- and postgraduate students of medicine, and postgraduate students of biology/biochemistry, chemistry and pharmacy. The graduates carry out research studies in vascular biology towards a doctoral degree (German MD or PhD). Based on an interdisciplinary approach the program offers students the opportunity to acquire a broad scientific knowledge in the biochemistry, molecular biology, physiology and cell biology of the vessel wall and the pathogenesis of vascular diseases as well the possibility to get trained in various methods of vascular cell biology, thereby receiving a broad education in this central biomedical research field.
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