GORDON CONFERENCE ON ATHEROSCLEROSIS 1997

描述（改编自研究者摘要）：本应用程序是 第13届戈登研究会议的部分支持， 运动会将在纽约梅里登的金博尔联盟学院举行 汉普郡，1997年6月15日至20日。 这次国际会议， 自1971年以来每两年举行一次，将提供一个最全面的概述， 令人兴奋的最新进展，血管生成，炎症，基质生物学， 和脂蛋白受体结构-功能，因为它们与动脉粥样硬化形成有关。 在发病机制中涉及的基本分子和结构的进展 将讨论动脉粥样硬化。 重点将放在以下方面的作用： 基因治疗在修改动脉病;和一个会议计划在 使用新的动物模型来研究这种疾病的病因。 九 计划的会议将包括：1。 血栓形成与 和动脉粥样硬化。 2. 遗传学方法研究 动脉粥样硬化 3. 基因转移技术在血管生成和 动脉粥样硬化 4. 炎症细胞在病变进展中的作用， 回归分析 5. 影响血管张力和生长的细胞信号事件。 6. 脂蛋白受体生物学与胆固醇的新进展 贩卖人口 7. 研究动脉粥样硬化病变的实验模型， 斑块破裂 8. 细胞外基质和重塑。 9. 新 研究人类遗传特征和动脉粥样硬化易感性的方法。 这次会议将成为新知识整合的主要工具 在动脉粥样硬化研究领域， 到会议结束时，与会者将有望收集到一个 更好地了解与之相关的生物过程 动脉粥样硬化 这次会议将包括妇女和少数民族作为会议 领导人和讨论者;来自欧洲的科学家也将参加， 会议领导人。 将尽一切努力，包括博士后 研究员和研究生参加了 下午;而且，他们中的一些人被要求在会后领导 讨论下午会议期间提出的材料。 它 这是戈登会议联合主席的严肃意图（D。 Hajjar和H.霍布斯），介绍新的和年轻的调查人员作为发言人， 他们在本次会议上发挥了领导作用。

DESCRIPTION (Adapted from the Investigator's Abstract): This application is for partial support of the 13th Gordon Research Conference on Atherosclerosis to be held at Kimball Union Academy in Meriden, New Hampshire on June 15-20, 1997. This international conference, which has been held every two years since 1971, will provide an overview of the most exciting recent developments in angiogenesis, inflammation, matrix biology, and lipoprotein receptor structure-function as they relate to atherogenesis. Both basic molecular and structural advances involved in the pathogenesis of atherosclerosis will be discussed. Emphasis will be placed on the role of gene therapy in modifying the arteriopathy; and a session is planned on the use of new animal models to study the etiology of this disease. Nine sessions are planned and will cover: 1. Molecular links between thrombosis and atherosclerosis. 2. Genetic approaches to the study of atherosclerosis. 3. Use of gene transfer techniques in angiogenesis and atherogenesis. 4. Role of inflammatory cells in lesion progression and regression. 5. Cell signaling events affecting vascular tone and growth. 6. New advances in lipoprotein receptor biology and cholesterol trafficking. 7. Experimental models to study atherosclerotic lesions and plaque rupture. 8. Extracellular matrix and remodeling. 9. New approaches to study human genetic traits and atherosclerosis susceptibility. This conference will be a major vehicle for the integration of new knowledge in the field of atherosclerosis research and it is planned in such a way that by the end of the conference, the participants will hopefully glean a better understanding of the biological processes associated with atherogenesis. This conference will include women and minorities as session leaders and discussants; scientists from Europe will also participate as session leaders. Every attempt will be made to include post-doctoral fellows and graduate students in the poster sessions scheduled in the afternoons; and, some of them have been asked to lead post-session discussions on the material presented during those afternoon sessions. It is the serious intention of the Co-chairs of this Gordon Conference (D. Hajjar and H. Hobbs) to profile new and young investigators as speakers in this Conference and for them to have a leadership role at this meeting.