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Could mitotic exhaustion of NG2-glia explain the remyelination deficit in MS?

Could mitotic exhaustion of NG2-glia explain the remyelination deficit in MS?
NG2-胶质细胞的有丝分裂耗竭能否解释多发性硬化症的髓鞘再生缺陷？
批准号：
414164
负责人：
Kokoeva Maia
金额：
$7.29万
依托单位：
Research Institute of the McGill University Health Centre
依托单位国家：
加拿大
项目类别：
Operating Grants
财政年份：
2019
资助国家：
加拿大
项目状态：
已结题
起止时间：
2019-09-01 至 2020-09-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=414164&lang=en
关键词：
Demyelination Multiple Sclerosis Myelin Ng2-Glia Oligodendrocyte Precursor Cells (Opcs) Remyelination

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中文摘要
翻译
多发性硬化症（MS）是一种包裹在神经元过程周围、帮助脑细胞之间交流的脂肪鞘受损的疾病。在疾病的初始阶段，大脑能够修复这些损伤，但是
英文摘要
Multiple sclerosis (MS) is a disease in which the fatty sheaths that are wrapped around neuronal processes and that aid communication between brain cells become damaged. During the initial phase of the disease, the brain is able to repair those damages bu
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