The Identity Vitality-Pathology model: A novel theoretical framework proposing "identity state" as a modulator of the pathways from structural to health inequity.
The Identity Vitality-Pathology model: A novel theoretical framework proposing "identity state" as a modulator of the pathways from structural to health inequity.
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身份活力-病理学模型:一种新颖的理论框架,提出“身份状态”作为从结构性不平等到健康不平等路径的调节器。
DOI:
10.1016/j.socscimed.2022.115495
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发表时间:
2022-12
影响因子:
5.4
通讯作者:
Bey, Ganga S.
中科院分区:
文献类型:
--
作者:
Bey, Ganga S.
A strong evidence base (Krieger, 2014; Marmot and Allen, 2014; Conrad and Barker, 2010; Adler and Stewart, 2010; Williams and Collins, 2001; Crocker and Major, 1989) offers important, while arguably, incomplete conceptualizations of the causal factors contributing to differences in specific health outcomes between groups defined by demographic characteristics such as socioeconomic position, race, ethnicity, and gender. Focusing on the magnitude of group differences in the prevalence or incidence of a specific disease outcome has yielded an incomplete assessment of the ways in which structural inequity breeds health inequity (Ward et al., 2019). Such counterfactual methods of measuring disparities necessitate the assumption of a normal or optimal health state, which has traditionally been based on outcomes observed among individuals occupying what many social theories (Turner and Avison, 2003; Krieger, 1990; Crocker and Major, 1989) consider the most privileged social position in the United States: White men. This approach does correctly assert that social disadvantage unjustly deprives individuals of access to optimal health states. However, the White male referent overlooks two important considerations: one, that many health-impacting processes are not comparable across social groups due to the interdependency of these processes with social group membership (Bey et al., 2016, 2018a, 2018b, 2019; Schwartz, 2017; Slavich and Irwin, 2014; Harnois and Ifatunji, 2011), and two, that unjust advantage can also be disruptive of optimal health (Bey et al., 2016; Fujishiro, 2009), an idea which challenges the notion of the White male as a model of health.Researchers have debated extensively on the challenge of establishing norms of health (Metzl, 2003, 2009; Amundson, 2000). The aim of this work is not to further engage this discussion but rather to contend that like illness resulting from relative deprivation, there are pathologies which arise from relative excess. Socially-constructed dominance and subordinance each confer vulnerability to specific diseases and resilience against others. A more accurate characterization of the mechanisms by which structural inequity acts to yield unequal health requires reconceptualizing notions of health and illness to be more inclusive of the often normalized symptoms of pathology exhibited by members of dominant-status groups.
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影响因子:
8.5
作者:
Batavia, Chelsea;Nelson, Michael Paul;Wallach, Arian D.
通讯作者:
Wallach, Arian D.
影响因子:
3.9
作者:
Crocker, J
通讯作者:
Crocker, J
影响因子:
5
作者:
Conrad, Peter;Barker, Kristin K.
通讯作者:
Barker, Kristin K.
影响因子:
4.7
作者:
Bey, G. S.;Jesdale, B.;Kiefe, C.
通讯作者:
Kiefe, C.
DOI:
10.1111/j.1749-6632.2009.05337.x
发表时间:
2010-01-01
期刊:
BIOLOGY OF DISADVANTAGE: SOCIOECONOMIC STATUS AND HEALTH
影响因子:
--
作者:
Adler, Nancy E.;Stewart, Judith
通讯作者:
Stewart, Judith