Depletion of Tissue-Specific Ion Transporters Causes Differential Expression of PRL Targets in Response to Increased Levels of Endogenous PRL

Depletion of Tissue-Specific Ion Transporters Causes Differential Expression of PRL Targets in Response to Increased Levels of Endogenous PRL
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组织特异性离子转运蛋白的消耗导致 PRL 靶标的差异表达,以响应内源性 PRL 水平的增加

DOI:
10.3389/fendo.2018.00683
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发表时间:
2018-11
影响因子:
5.2
通讯作者:
Yin Zhan
Yin Zhan
中科院分区:
医学2区
文献类型:
--
作者:
Shu Tingting;Shu Yuqin;Gao Yanping;Jin Xia;He Jiangyan;Zhai Gang;Yin Zhan

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Prolactin (PRL) has been considered a key regulator of ion uptake in zebrafish. The genes slc12a10.2 and slc12a3, which are Na+ and chloride Cl− co-transporters, have been reported to be regulated by PRL in freshwater fish. The integrative network of PRL
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