Magnetic resonance imaging of the functional anatomy of the inferior rectus muscle in superior oblique muscle palsy.
Magnetic resonance imaging of the functional anatomy of the inferior rectus muscle in superior oblique muscle palsy.
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DOI:
10.1016/j.ophtha.2008.04.040
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发表时间:
2008-11
期刊:
影响因子:
13.7
通讯作者:
Demer JL
中科院分区:
文献类型:
--
作者:
Jiang L;Demer JL
Biomechanical modeling consistently indicates that superior oblique (SO) weakness alone is insufficient to explain the large hypertropia often observed in SO palsy. Magnetic resonance imaging (MRI) was used to investigate if any size or contractility changes in the inferior rectus (IR) may contribute. Prospective, case control study. 17 patients with unilateral SO palsy, and 18 orthotropic subjects. Surface coils were used to obtain sets of contiguous, 2 mm thick high-resolution, coronal MRI in different gazes. Cross sectional areas of the IR and SO muscles were determined in supraduction and infraduction for evaluation of size and contractility. Diagnosis of SO palsy was based on clinical presentations, and subnormal contractility and SO size less than the normal 95% confidence limit. Cross sectional areas of the IR and SO muscles. Patients had 15.9 ± 7.2 (mean ± standard deviation) Δ central gaze hypertropia and exhibited ipsilesional SO atrophy and subnormal contractility. Mean ipsilesional, contralesional and normal IR cross-sections were 28.5 ± 3.5 mm2, 31.9 ± 3.8 mm2 and 31.8 ± 5.8 mm2, while mean contractility was 16.5 ± 3.8 mm2, 20.5 ± 4.1 mm2, and 16.6 ± 4.8 mm2, respectively. Ipsilesional IR cross-sections and contractility were significantly less than contralesional (P < 0.01). In SO palsy, the contralesional IR is larger and more contractile than the ipsilesional IR, reflecting likely neurally mediated changes that augment the relatively small hypertropia due to SO weakness alone. Recession of the hyperfunctioning contralesional IR recession in SO palsy is a physiologic therapy.
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影响因子:
1.8
作者:
ROBINSON, DA
通讯作者:
ROBINSON, DA
影响因子:
1.6
作者:
Clark, RA;Rosenbaum, AL;Demer, JL
通讯作者:
Demer, JL
影响因子:
--
作者:
ELLIS F D;HELVESTON E M
通讯作者:
HELVESTON E M
DOI:
10.1016/0010-4809(84)90012-0
发表时间:
1984-01-01
期刊:
COMPUTERS AND BIOMEDICAL RESEARCH
影响因子:
--
作者:
MILLER, JM;ROBINSON, DA
通讯作者:
ROBINSON, DA
影响因子:
4.4
作者:
Guyton, David L.
通讯作者:
Guyton, David L.