Subacute Ingestion of Caffeine and Oolong Tea Increases Fat Oxidation without Affecting Energy Expenditure and Sleep Architecture: A Randomized, Placebo-Controlled, Double-Blinded Cross-Over Trial.
Subacute Ingestion of Caffeine and Oolong Tea Increases Fat Oxidation without Affecting Energy Expenditure and Sleep Architecture: A Randomized, Placebo-Controlled, Double-Blinded Cross-Over Trial.
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亚急性摄入咖啡因和乌龙茶增加脂肪氧化而不影响能量消耗和睡眠结构:一项随机,安慰剂对照,双盲交叉试验。
DOI:
10.3390/nu12123671
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发表时间:
2020-11-28
期刊:
影响因子:
5.9
通讯作者:
Tokuyama K
中科院分区:
文献类型:
--
作者:
Zhang S;Takano J;Murayama N;Tominaga M;Abe T;Park I;Seol J;Ishihara A;Tanaka Y;Yajima K;Suzuki Y;Suzuki C;Fukusumi S;Yanagisawa M;Kokubo T;Tokuyama K
Ingesting oolong tea or caffeine acutely increases energy expenditure, and oolong tea, but not caffeine, stimulates fat oxidation. The acute effects of caffeine, such as increased heart rate and interference with sleep, diminish over 1–4 days, known as caffeine tolerance. During each 14-day session of the present study, 12 non-obese males consumed oolong tea (100 mg caffeine, 21.4 mg gallic acid, 97 mg catechins and 125 mg polymerized polyphenol), caffeine (100 mg), or placebo at breakfast and lunch. On day 14 of each session, 24-h indirect calorimetry and polysomnographic sleep recording were performed. Caffeine and oolong tea increased fat oxidation by ~20% without affecting energy expenditure over 24-h. The decrease in the respiratory quotient by oolong tea was greater than that by caffeine during sleep. The effect of oolong tea on fat oxidation was salient in the post-absorptive state. These findings suggest a role of unidentified ingredients in oolong tea to stimulate fat oxidation, and this effect is partially suppressed in a postprandial state. Two weeks of caffeine or oolong tea ingestion increased fat oxidation without interfering with sleep. The effects of subacute ingestion of caffeine and oolong tea differed from the acute effects, which is a particularly important consideration regarding habitual tea consumption.
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影响因子:
7.1
作者:
DULLOO, AG;GEISSLER, CA;MILLER, DS
通讯作者:
MILLER, DS
影响因子:
3.1
作者:
Liu, Pan-Pan;Yin, Jun-Feng;Xu, Yong-Quan
通讯作者:
Xu, Yong-Quan
影响因子:
8.9
作者:
Nielsen, L. S.;Danielsen, K. V.;Sorensen, T. I. A.
通讯作者:
Sorensen, T. I. A.
DOI:
10.1523/jneurosci.6730-10.2011
发表时间:
2011-07-06
期刊:
The Journal of neuroscience : the official journal of the Society for Neuroscience
影响因子:
--
作者:
Lazarus M;Shen HY;Cherasse Y;Qu WM;Huang ZL;Bass CE;Winsky-Sommerer R;Semba K;Fredholm BB;Boison D;Hayaishi O;Urade Y;Chen JF
通讯作者:
Chen JF
影响因子:
6.1
作者:
Nakai, M;Fukui, Y;Kiso, Y
通讯作者:
Kiso, Y