Leaf senescence: gene expression and regulation.
Leaf senescence: gene expression and regulation.
复制标题
叶子衰老:基因表达和调控。
DOI:
10.1007/978-1-4615-5925-2_12
复制
发表时间:
1997
影响因子:
6.4
通讯作者:
R. Amasino
中科院分区:
文献类型:
--
作者:
L. M. Weaver;E. Himelblau;R. Amasino
Each autumn leaf senescence leaves its mark on the planet in the form of dramatic changes in color that can be seen from space. Annually, leaf senescence mediates the breakdown of 300 million tons of chlorophyll while changing green forests and fields to yellow and orange (1). The drama of these color changes is matched by the dramatic nature of the cellular events that underlie them. In 1961, Leopold (2) described leaf senescence as “distinctive changes in morphology, pigmentation, and internal nutrition of the plant” with “positive ecological or physiological value.” The benefit to the plant is the reclamation of nitrogen and other nutrients from leaves that have reached the end of their photosynthetic productivity. This reclamation entails the breakdown of the chloroplasts, the conversion of breakdown products into translocatable molecules and the movement of those molecules from the senescing leaf to growing regions of the plant, including the younger leaves and developing fruits. Together, these events are known as the “senescence syndrome.” Currently, numerous laboratories are expanding the definition of the senescence syndrome to encompass the molecular and genetic components of this important process. Leaf senescence is now understood to be a nuclear-regulated, developmental pathway facilitating the recovery of nutrients from spent leaves.
影响因子:
11.6
作者:
HENSEL, LL;GRBIC, V;BLEECKER, AB
通讯作者:
BLEECKER, AB