Podocyte Activation of NLRP3 Inflammasomes Contributes to the Development of Proteinuria in Lupus Nephritis.

Podocyte Activation of NLRP3 Inflammasomes Contributes to the Development of Proteinuria in Lupus Nephritis.
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NLRP3 炎症小体的足细胞激活导致狼疮性肾炎蛋白尿的发生

DOI:
10.1002/art.40155
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发表时间:
2017-08
期刊:
Arthritis & rheumatology (Hoboken, N.J.)
影响因子:
--
通讯作者:
Fu SM
Fu SM
中科院分区:
其他
文献类型:
--
作者:
Fu R;Guo C;Wang S;Huang Y;Jin O;Hu H;Chen J;Xu B;Zhou M;Zhao J;Sung SJ;Wang H;Gaskin F;Yang N;Fu SM

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狼疮性肾炎(LN)蛋白尿的发生与足细胞功能障碍有关。NLRP3炎性体与LN的发病机制有关。本研究的目的是调查是否NLRP 3炎性体激活参与足细胞损伤的发展在LN。
Development of proteinuria in lupus nephritis (LN) is associated with podocyte dysfunction. The NLRP3 inflammasome has been implicated in the pathogenesis of LN. The purpose of this study was to investigate whether NLRP3 inflammasome activation is involved in the development of podocyte injury in LN.
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