Relationships between catastrophic thought, bodily sensations and physical symptoms.
Relationships between catastrophic thought, bodily sensations and physical symptoms.
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DOI：
10.1186/s13030-017-0110-z
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发表时间：
2017
影响因子：
2.1
通讯作者：
Nakao M
中科院分区：
文献类型：
--
作者：
Seto H;Nakao M
研究人员最近开始寻找没有明显生物原因的身体症状的认知解释。诸如躯体化、躯体感觉放大和躯体感觉灾变等概念被用来解释这些现象。当这些身体感觉变得更强烈和更痛苦时，体感就会放大。躯体感觉灾害化是将这些身体感觉归因于无法忍受的功能调节或作为严重疾病的迹象的倾向。这会导致患者过度关注这些身体感觉。然而，目前还没有评估躯体感觉灾变的标准，也没有标准的诊断标准。这项研究有两个目标：建立一个评估躯体感觉灾变的量表，并调查躯体感觉放大、躯体感觉灾变和身体症状之间的关系。在这项研究的第一部分，我们制定了量表，有231名学生参与，平均年龄为20.1百万岁。其中，57%的参与者是女性。在研究的第二部分，有两组参与者。第一组由158名非患者受试者组成，其中56%为女性，平均年龄20.2岁。第二组有33例躯体形式障碍患者接受门诊治疗。这些参与者的平均年龄为48.8岁，其中67%是女性。这项研究的第二部分是使用标准化的自我评级问卷来评估躯体感觉放大和灾难。我们开发了一个由27个条目组成的量表，我们称之为躯体感觉灾变量表(SSCS)。SSCS评估了五个关键领域，我们的分析证实它是有效的和高度可靠的。该量表表明，研究第二部分的患者组在躯体感觉放大和灾难方面的得分都高于对照组。此外，协方差结构分析的结果显示，通过“躯体感觉灾变”形成的“躯体感觉放大”形式与“身体症状”之间存在显著的因果关系。这种关系在两组参与者中都存在。患者组和非患者组之间的关键区别在于，躯体感觉灾难对患者组参与者的身体症状有更大的影响。在这项研究中，我们开发了SSCS，它使我们能够经验性地测量躯体感觉灾难。然后，我们阐明了躯体感觉放大、躯体感觉灾变和身体症状之间的关系。在未来，我们希望能够将我们的新理解应用于开发干预技术，以缓解由躯体感觉灾难引起的身体症状。本文的在线版本(doi：10.1186/s13030-0170110-z)包含补充材料，授权用户可以使用。
Researchers have recently begun to seek cognitive explanations for physical symptoms with no obvious biological cause. Concepts such as somatization, somatosensory amplification, and somatosensory catastrophizing have been invoked to explain these phenomena. Somatosensory amplification occurs when these bodily sensations become stronger and more painful. Somatosensory catastrophizing is the tendency to attribute these bodily sensations to unbearable functional modulation or as signs of serious illness. This causes the sufferer to pay excessive attention to these physical sensations. However, there is no scale for evaluating somatosensory catastrophizing, and there are no standard diagnostic criteria. There were two objectives for this study: to develop a scale for evaluating somatosensory catastrophizing and to investigate relationships between somatosensory amplification, somatosensory catastrophizing, and physical symptoms. In the first part of this study, in which we developed the scale, there were 231 student participants with an average age of 20.1 years. Of these, 57% of the participants were female. In the second part of the study, there were two groups of participants. The first group consisted of 158 non-patient subjects, 56% of whom were female, with an average age of 20.2 years. There were 33 outpatients receiving treatment for somatoform disorders in the second group. The average age of these participants, of whom 67% were female, was 48.8 years. The second part of the study was conducted using standardized self-rating questionnaires to assess somatosensory amplification and catastrophizing. We developed a 27-item scale, which we have called the Somatosensory catastrophizing scale (SSCS). The SSCS assesses five key areas, and our analysis confirmed it to be valid and highly reliable. The scale identified that the patient group from the second part of the study scored more highly than the control group for both somatosensory amplification and catastrophizing. Additionally, the results of covariance structure analyses revealed a significant causal relationship of the form “somatosensory amplifcation” via “somatosensory catastrophizing” to “physical symptoms”. This relationship held in both groups of participants. The key difference between the patient and non-patient groups was that somatosensory catastrophizing had a greater impact on the physical symptoms of the participants in the patient group. In this study, we developed the SSCS, which enables us to measure somatosensory catastrophizing empirically. We then clarified the relationship between somatosensory amplification, somatosensory catastrophizing, and physical symptoms. In the future, we expect to be able to apply our new understanding to developing intervention techniques to mitigate the physical symptoms caused by somatosensory catastrophizing. The online version of this article (doi:10.1186/s13030-017-0110-z) contains supplementary material, which is available to authorized users.
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影响因子：
2.3
作者：
Nakao, M;Myers, P;Benson, H
通讯作者：
Benson, H
影响因子：
6.9
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影响因子：
3
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影响因子：
3
作者：
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影响因子：
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作者：
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通讯作者：
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