Inhibition of EPAC2 Attenuates Intracerebral Hemorrhage-Induced Secondary Brain Injury via the p38/BIM/Caspase-3 Pathway

Inhibition of EPAC2 Attenuates Intracerebral Hemorrhage-Induced Secondary Brain Injury via the p38/BIM/Caspase-3 Pathway
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抑制 EPAC2 通过 p38/BIM/Caspase-3 途径减轻脑出血引起的继发性脑损伤

DOI:
10.1007/s12031-018-1215-y
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发表时间:
2019-01
影响因子:
3.1
通讯作者:
Chen Gang
Chen Gang
中科院分区:
医学4区
文献类型:
--
作者:
Zhuang Yan;Xu Hui;Richard Seidu A;Cao Jie;Li Haiying;Shen Haitao;Yu Zhengquan;Zhang Jian;Wang Zhong;Li Xiang;Chen Gang

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Exchange proteins directly activated by cAMP (EPACs) are critical cAMP-dependent signaling pathway intermediaries that have been implicated in the pathogenesis of several human diseases, particularly neurological disorders. However, their pathogenic role
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