Microcirculation of the alimentary tract: II. Pathophysiology of edema
Microcirculation of the alimentary tract: II. Pathophysiology of edema
复制标题
消化道的微循环:II。
DOI:
10.1016/0016-5085(83)90209-3
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发表时间:
1983
期刊:
影响因子:
29.4
通讯作者:
J. Barrowman
中科院分区:
文献类型:
--
作者:
D. Granger;J. Barrowman
Recognition and assessment of edema of gastrointestinal organs is difficult. While resection or biopsy specimens are commonly reported to show edema in association with a wide variety of pathologic processes, the clinician has few methods for detecting and quantifying edema of the gut in patients. Occasionally, endoscopic appearances of the stomach, duodenum, or colon may suggest the presence of mucosal edema. Radiologic appearances of intestinal edema on barium examination are welldescribed (1). Where colloid osmotic pressure disturbances are the cause (Figure l), diffuse intestinal edema can usually be demonstrated radiologically when the serum albumin is~‘2 g/l00 ml (2). Radiologic recognition of bowel edema has also been described in inflammatory diseases (3-5) and allergic conditions(6-8). A common cause of radiologically demonstrable gastrointestinal edema is cirrhosis; most readily shown are diffuse changes throughout the small intestine with thickening of mucosal folds and disorganization of peristaltic contractions. Although it is difficult to demonstrate mucosal edema of the stomach, some cases of cirrhosis with small bowel edema will show thickening of gastric rugae. In the colon, reduced distensibility and deformation of the haustral pattern indicate edematous changes, which are most often confined
DOI:
10.1152/ajpheart.1982.242.4.h557
发表时间:
1982
期刊:
The American journal of physiology
影响因子:
--
作者:
Dillon,BC;Saba,TM
通讯作者:
Saba,TM