Biological Theories of Violence
Biological Theories of Violence
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暴力的生物学理论
DOI：
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发表时间：
1991
期刊：
影响因子：
--
通讯作者：
A. Scerbo
中科院分区：
文献类型：
--
作者：
A. Raine;A. Scerbo
本章描述了一些暴力的生物学理论，以便为后面关于攻击的神经心理学的章节提供一个一般的框架。这里描述的理论选择(额叶功能障碍、左半球功能障碍、左额-颞叶-边缘损伤、语言偏侧化减少、觉醒不足、迷走神经痉挛和胎儿神经发育障碍)绝不是详尽的。之所以选择理论，是因为它们代表了关于暴力的重要生物学观点，并得到了相当广泛的研究(例如，额叶理论)，因为它们可能引起神经心理学家的特别兴趣(减少了语言的偏侧化)，或者因为它们代表了相对较新的观点，这些观点没有得到充分的研究，但代表了未来研究的有希望的路线(例如胎儿发育不良、迷走神经痉挛)。特别是，基于类固醇、多巴胺、去甲肾上腺素、5-羟色胺、GABA/苯二氮卓类受体的研究以及动物攻击性的神经解剖学的暴力理论没有包括在内，因为它们可能与研究人类暴力的神经心理学家不那么直接相关；然而，它们是重要的生物学视角，在对暴力的生物学基础进行任何完整解释时都应予以考虑。
This chapter describes a number of biological theories of violence in order to provide a general framework for the following chapters on the neuropsychology of aggression. The selection of theories described here (frontal dysfunction, left hemisphere dysfunction, left fronto-temporal-limbic damage, reduced lateralization for language, underarousal, vagotonia, and fetal neural maldevelopment) are by no means exhaustive. Theories have been chosen because they represent important biological perspectives on violence and have been fairly extensively researched (e.g., frontal theory), because they may be of special interest to neuropsychologists (reduced lateralization for language), or because they represent relatively new perspectives that have been under-researched but that represent promising lines for future study (e.g., fetal maldevelopment, vagotonia). In particular, theories of violence based on research on steroids, dopamine, norepinephrine, serotonin, GABA/benzodiazepine receptors, and the neuroanatomy of aggression in animals have not been included, since they may be of less direct interest to neuropsychologists researching violence in humans; they are nevertheless important biological perspectives that should be taken into account in any complete explanation of the biological basis of violence.
影响因子：
3.7
作者：
RAINE, A;REYNOLDS, GP;SHEARD, C
通讯作者：
SHEARD, C