Co-targeting Aurora kinase A and Bcl-2 synergistically inhibits the viability in double-hit lymphoma cells

Co-targeting Aurora kinase A and Bcl-2 synergistically inhibits the viability in double-hit lymphoma cells
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共同靶向 Aurora 激酶 A 和 Bcl-2 协同抑制双重打击淋巴瘤细胞的活力

DOI:
10.21037/tcr.2017.06.41
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发表时间:
2017-08
影响因子:
0.9
通讯作者:
Zhang Hui-Lai
Zhang Hui-Lai
中科院分区:
医学4区
文献类型:
--
作者:
Kong Ling-Zhe;Jia Xiao-Hui;Song Zheng;Qiu Li-Hua;Li Lan-Fang;Qian Zheng-Zi;Zhou Shi-Yong;Liu Xian-Ming;Ren Xiu-Bao;Meng Bin;Fu Kai;Wang Ping;Wang Xian-Huo;Zhang Hui-Lai

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背景:双重打击淋巴瘤(DHL)是一种罕见的高度恶性B细胞淋巴瘤,以MYC和Bcl-2或Bcl-6基因易位为特征。DHL的治疗仍然是一个重大的临床挑战,由于非常不理想的结果。创新药
Background: Double-hit lymphoma (DHL) is a rare high-grade B-cell lymphoma characterized by MYC and Bcl-2 or Bcl-6 gene translocations. The treatment of DHL remains a substantial clinical challenge due to remarkably undesirable outcomes. Innovative drugs
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