Molecular mechanisms of neurodegeneration in Parkinson's disease

帕金森病神经退行性变的分子机制

• 批准号: 316310
• 负责人: Kalia Lorraine V
• 金额: $ 21.86万
• 依托单位: University of Toronto
• 依托单位国家: 加拿大
• 项目类别: Salary Programs
• 财政年份: 2014
• 资助国家: 加拿大
• 起止时间: 2014-10-01 至 2017-10-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=316310&lang=en
• 关键词: Alpha-Synuclein; Animal Models (C. Elegans); Animal Models (Rodent); Cell Culture Models; Drug Screens; Immunohistochemistry; Molecular Biology; Molecular Mechanisms Of Disease; Protein Biochemistry; Proteostasis

Parkinson's disease is a disabling neurological disorder that is increasing in prevalence and thus having a growing impact on the Canadian population. This disease results from a process called neurodegeneration in which vulnerable nerve cells in the brai

帕金森氏病是一种致残性神经系统疾病，患病率正在增加，因此对加拿大人口的影响越来越大。这种疾病是由一种叫做神经变性的过程引起的，在这种过程中，大脑中脆弱的神经细胞