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The interplay of apoptosis proteins, mitochondria and premature senescence in beta-cell fate and diabetes

The interplay of apoptosis proteins, mitochondria and premature senescence in beta-cell fate and diabetes
细胞凋亡蛋白、线粒体和早衰在β细胞命运和糖尿病中的相互作用
批准号：
450487
负责人：
Luciani Dan S
金额：
$61.02万
依托单位：
BC Children's Hospital Research Institute
依托单位国家：
加拿大
项目类别：
Operating Grants
财政年份：
2021
资助国家：
加拿大
项目状态：
未结题
起止时间：
2021-09-01 至 2026-09-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=450487&lang=en
关键词：
Apoptosis Autophagy / Mitophagy Bcl-2 Family Proteins Beta-Cell Biology Cellular Stress Diabetes High Content Imaging Islet Biology Mitochondrial Morphology And Function Senescence

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Diabetes is a serious disease in which high levels of blood sugar (glucose) damage the body and markedly shortens life-span. Glucose is normally kept low by the hormone insulin, which is released from beta-cells in the pancreas. Diabetes develops, in larg
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Control of beta-cell mitochondrial physiology and adaptation by core anti-apoptosis proteins
Control of beta-cell mitochondrial physiology by core anti-apoptosis proteins