Characterization of an mTORC1-independent B cell differentiation program
Characterization of an mTORC1-independent B cell differentiation program
批准号:
25670236
负责人:
MATSUDA Satoshi
金额:
$2.5万
依托单位国家:
日本
项目类别:
Grant-in-Aid for Challenging Exploratory Research
财政年份:
2013
资助国家:
日本
项目状态:
已结题
起止时间:
2013-04-01 至 2015-03-31
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期刊论文(7)
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B-lineage specific loss of mTORC1 signal causes selective production of IgA against commensal bacteria.
B 谱系特异性 mTORC1 信号缺失会导致针对共生细菌选择性产生 IgA。
DOI:
--
发表时间:
2014
期刊:
影响因子:
--
作者:
[Ohtani, M., Fujii, H., Ohara, O., Koyasu, S., Kubo, M., and Matsuda, S.]
通讯作者:
S.
mTORC1シグナルによるB細胞分化制御
mTORC1 信号控制 B 细胞分化
DOI:
--
发表时间:
2014
期刊:
影响因子:
--
作者:
[松田達志, 大谷真志, 小安重夫,久保允人, 平尾敦]
通讯作者:
平尾敦
mTOR complex 1 is critical for B cell development but not IgA production.
mTOR 复合物 1 对 B 细胞发育至关重要,但对 IgA 产生则不然。
DOI:
--
发表时间:
2013
期刊:
影响因子:
--
作者:
[Ohatni, M., Fujii, H., Koyasu, S., Kubo, M., and Matsuda, S.]
通讯作者:
S.
関西医科大学附属生命医学研究所生体情報部門ホームページ
关西医科大学生物医学科学研究所生物信息学部主页
DOI:
--
发表时间:
期刊:
影响因子:
--
作者:
[]
通讯作者:
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Immune regulation through dendritic cell-specific mTOR pathway
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Frictional Properties without lubricant and structure of diamond. like-carbon coatings
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Stochastic Random Input Ground Motion Model and its Applications
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The Correlation among the Repertoires in the Performances of Operas at the Italian Cities and Vienna
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Regulation of dendritic cell activation through the PI3K-p38 MAPK
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超対称弦ソリトンと超共形代数の表現
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Representation Theories of Superconformal Algebras and Superstring
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依托单位:
Renormalization Group and Migdal-Kadanoff Transformation in Gauge and Spin Systems
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依托单位: