Dissection of CrRLK1L signalling pathways regulating disease susceptibility and resistance to powdery mildew (B09)

本项目研究了拟南芥CrRLK1L费洛尼亚及其Ralf多肽配体支持白粉病侵染的分子机制。额外的CrRLK1基因通过一个基因家族的感病和抗病因子影响白粉病的侵染。本项目将研究拟南芥CrRLK1L和Ralf多样性在白粉病入侵过程中的不同调控功能，以及它们与阿魏酸菌的遗传和生化相互作用。

This project examines the molecular mechanisms of the Arabidopsis CrRLK1L FERONIA, together with its RALF peptide ligands, supporting powdery mildew infection. Additional CrRLK1Ls affect powdery mildew invasion with susceptibility and resistance factors within one gene family. This project will study Arabidopsis CrRLK1L and RALF diversity underlying differential regulatory functions during powdery mildew invasion and their genetic and biochemical interplay with FERONIA.