Analysis of cauda equina compression in lumobar spinal stenosis

腰椎管狭窄症马尾受压分析

• 批准号: 11671451
• 负责人: TAKAHASHI Keisuke
• 金额: $ 2.11万
• 依托单位: Saitama Medical School
• 依托单位国家: 日本
• 项目类别: Grant-in-Aid for Scientific Research (C)
• 财政年份: 1999
• 资助国家: 日本
• 起止时间: 1999 至 2001
• 项目状态: 已结题
• 来源: https://kaken.nii.ac.jp/grant/KAKENHI-PROJECT-11671451/
• 关键词: lumbar apine; spinal stenosis; intermittent claudication; epidural pressure; the elderly; posture; walking; exercise; 圧迫力; 腰部脊柱管狭窄症; 神経圧迫動態; 圧測定

The study of epidural pressure measurements clearly demonstrated that the local extradural pressure at the stenotic level was changed by motion in patients with spinal stenosis. The pressure was low in lying and sitting, and high in standing. Especially, the pressure was increased by lumbar extension, but decreased by forward flexion. Changes of the epidural pressure correlated with the occurence of clinical symptoms of nerve roots compression. These pressure changes may explain the postural dependency of symptoms in spinal stenosisThe epidural pressure was also changed at walking in patients with neurogenic intermittent claudication. The epidural pressure change had a pattern of increase and decrease, and this pattern was repeated during walking. The epidural pressure was high at simple walking and low at walking with lumbar flexion. Intermittent compression to the nerve roots during walking might have a important role in the pathogenesis of neurogenic intermittent claudicationThe pressure was extremely increased by trunk muscle exercises e. g. back muscle strength exercise. This result imply that back muscle exercise might be dangerous for patients with lumbar spinal stenosisIn conclusion, static compression in lumbar spinal stenosis is low compared to lumbar disc herniation. Dynamic compression by lumbar motion would be important for the pathophysiology of lumbar spinal stenosis

硬膜外压力测量的研究清楚地表明，脊柱狭窄患者的运动改变了狭窄水平的局部局部压力。撒谎和坐着的压力很低，站立高。特别是，腰部伸展会增加压力，但通过正向屈曲降低。硬膜外压的变化与神经根部压缩的临床症状的发生相关。这些压力的变化可能解释了脊柱狭窄中症状的姿势依赖性，在神经源性间歇性lau不平患者中行走时，硬膜外压力也发生了变化。硬膜外压力的变化具有增加和减少的模式，并且在步行过程中重复了这种模式。硬膜外压力在简单行走时高，并且在带着腰部屈曲时步行低。步行过程中对神经根的间歇性压缩可能在神经发生的间歇性clauraudication的发病机理中起重要作用，而躯干肌肉运动极大地增加了压力。 g。背部肌肉力量运动。该结果表明，背部肌肉运动可能对腰椎狭窄的患者结论可能是危险的，与腰椎间盘突出症相比，腰椎狭窄的静态压缩较低。腰运动的动态压缩对于腰椎狭窄的病理生理非常重要

项目成果

高橋啓介ほか: "腰部脊柱管狭窄症の病態、馬尾の機能的圧迫から"現代医療. 33. 23-27 (2001)

Keisuke Takahashi 等人：“马尾功能性压迫引起的腰椎管狭窄的病理学”现代医学。 33. 23-27 (2001)

高橋啓介ほか: "硬膜外圧からみた馬尾性間欠跛行"骨・関節・靭帯. 12(4). 377-381 (1999)

Keisuke Takahashi 等人：“从硬膜外压力观察到的马尾间歇性跛行”骨骼、关节和韧带 12(4) 377-381 (1999)。

Keisuke Takahashi, et al: "Nerve root presure in lumbar disc herniation"Spine. 24(19). 2003-2006 (1999)

Keisuke Takahashi 等人：“腰椎间盘突出症中的神经根压力”脊柱。

Keisuke Takahashi, et al: "Dynamics of cauda equina compression in lumber spinal stenosis"Mechanical Loading of Bones and Joints. Springer. 237-243 (1999)

Keisuke Takahashi 等人：“腰椎管狭窄症中马尾受压的动力学”骨骼和关节的机械负载。

Keisuke Takahashi: "Cauda equina intermittent claudication in view of the epidural pressue"Kotu-Kansetu-Jintai. 12(4). 377-381 (1999)

Keisuke Takahashi：“由于硬膜外压力导致马尾间歇性跛行”Kotu-Kansetu-Jintai。