Cytokine expression in the cochlear tissue in the course of wound healing caused by acoustic trauma

声损伤伤口愈合过程中耳蜗组织细胞因子的表达

• 批准号: 19791197
• 负责人: SATO Kunihiro
• 金额: $ 1.34万
• 依托单位: Niigata University
• 依托单位国家: 日本
• 项目类别: Grant-in-Aid for Young Scientists (B)
• 财政年份: 2007
• 资助国家: 日本
• 起止时间: 2007 至 2009
• 项目状态: 已结题
• 来源: https://kaken.nii.ac.jp/grant/KAKENHI-PROJECT-19791197/
• 关键词: 蝸牛; 音響外傷; 創傷治癒; マクロファージ; サイトカイン

Cochlea following acoustic trauma showed tissue damage leading to the wound healing with leukocyte infiltration. However, the amount of the leukocyte was not much. Interleukin-6, one of the important proinflammatory cytokines, was not expressed in the damaged cochlea.

声损伤后的耳蜗显示出组织损伤，导致伤口愈合并伴有白细胞浸润。但白细胞的数量并不多。白细胞介素 6 (Interleukin-6) 是重要的促炎细胞因子之一，在受损的耳蜗中不表达。