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Molecular mechanism of adhesion and deadhesion of endothelial cells required for angiogenesis

Molecular mechanism of adhesion and deadhesion of endothelial cells required for angiogenesis
血管生成所需内皮细胞粘附和死粘附的分子机制
批准号:
20370083
负责人:
MOCHIZUKI Naoki
金额:
$13.31万
依托单位国家:
日本
项目类别:
Grant-in-Aid for Scientific Research (B)
财政年份:
2008
资助国家:
日本
项目状态:
已结题
起止时间:
2008 至 2010

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中文摘要
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英文摘要
Angiopopoetin-1(Ang1)-Tie2 signal promotes the expression of Kruppel-like factor 2 that is known to be a transcription factor essential for vascular quiescence. In addition, Ang1/Tie2 induces Dll4 expression by inhibiting degradation of beta-catenin in a manner dependent on AKT. Dll4/Notch signal upon cell-cell contacts is believed to inhibit VEGF-VEGFR2 signal. Collectively, Ang1/Tie2 signal stabilizes endothelial cells by inhibiting VEGF-VEGFR2 signal.
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DOI: 10.1161/circresaha.107.170191
发表时间: 2008-02
期刊: Circulation research
影响因子: 20.1
作者: [N. Mochizuki;Young-Guen Kwon]
通讯作者: N. Mochizuki;Young-Guen Kwon
PSFl, a DNA replication factor expressed widely in stem and progenitor cells, drives tumorigenic and metastatic properties.
PSF1是在干细胞和祖细胞中广泛表达的DNA复制因子,驱动致瘤和转移特性。
DOI: --
发表时间: 2010
期刊: Cancer Res. 70
影响因子: --
作者: [Nagahama Y, Ueno M, Miyamoto S, Morii E, Minami T, Mochizuki N, Saya H, Takakura N.]
通讯作者: Takakura N.
A ngiopoietin-1/Tie2- mediated vascular maturation and quiescence are reregulate by Dll4-Notch signaling.
Ngiopoietin-1/Tie2 介导的血管成熟和静止由 Dll4-Notch 信号传导重新调节。
DOI: --
发表时间: 2010
期刊:
影响因子: --
作者: [長沼孝雄, 佐々木保典, 中川真一, 廣瀬哲郎, 三好知一郎, Naoki Mochizuki]
通讯作者: Naoki Mochizuki
DOI: 10.1158/0008-5472.can-09-3662
发表时间: 2010-02-01
期刊: CANCER RESEARCH
影响因子: 11.2
作者: [Nagahama, Yumi, Ueno, Masaya, Takakura, Nobuyuki]
通讯作者: Takakura, Nobuyuki
17
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    海外基金