MU-Opioid Receptor in Stress Induced Immunosupression

MU-阿片受体在应激诱导的免疫抑制中的作用

• 批准号: 7513850
• 负责人: Sabita Roy
• 金额: $ 10.98万
• 依托单位: UNIVERSITY OF MINNESOTA
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-05-01 至 2008-04-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7513850
• 关键词: AODR mortality; Adenylate Cyclase; Adrenalectomy; Alcohol abuse; Animals; Antiviral Agents; Area; Attenuated; Autoimmunity; Behavioral; Biological; CD4 Positive T Lymphocytes; Cell Differentiation process; Cellular Immunity; Chronic; Chronic stress; Clinical; Clinical Research; Communicable Diseases; Corticosterone; Corticotropin; Cyclic AMP; Data; Development; Dexamethasone; Disease; Effector Cell; Endorphins; Environment; Equilibrium; HPSE gene; Harvest; Helper-Inducer T-Lymphocyte; Human; Hydrocortisone; Immune; Immune response; Immune system; Immunity; Immunosuppression; Impairment; In Vitro; Individual; Infection; Infusion procedures; Interleukin-2; Interleukin-4; Interleukin-5; Investigation; Knockout Mice; Life Experience; Life Style; Link; Mediating; Mediator of activation protein; Modeling; Mus; Naloxone; Numbers; Opioid Peptide; Opioid Receptor; Opportunistic Infections; Outcome; Pathologic Processes; Phenotype; Play; Pneumococcal Pneumonia; Population; Predisposition; Process; Production; Protein Biosynthesis; Psychosocial Factor; Regulation; Risk Factors; Role; Specificity; Splenocyte; Stress; Substance abuse problem; Therapeutic; Therapeutic immunosuppression; Thinking; Time; Vaccination; Vaccines; Virus Diseases; Wild Type Mouse; beta-Endorphin; biological adaptation to stress; cytokine; design; emotional distress; endogenous opioids; human disease; hypothalamic-pituitary-adrenal axis; immune function; in vivo Model; mouse model; mu opioid receptors; neurochemistry; pathogen; programs; psychologic; receptor; response; restraint stress; role model; stressor; transcription factor; tumor