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MU-Opioid Receptor in Stress Induced Immunosupression

MU-Opioid Receptor in Stress Induced Immunosupression
MU-阿片受体在应激诱导的免疫抑制中的作用
批准号：
7513850
负责人：
Sabita Roy
金额：
$10.98万
依托单位：
UNIVERSITY OF MINNESOTA
依托单位国家：
美国
项目类别：
财政年份：
2007
资助国家：
美国
项目状态：
已结题
起止时间：
2007-05-01 至 2008-04-30
来源：
https://reporter.nih.gov/project-details/7513850
关键词：
AODR mortalityAdenylate CyclaseAdrenalectomyAlcohol abuseAnimalsAntiviral AgentsAreaAttenuatedAutoimmunityBehavioralBiologicalCD4 Positive T LymphocytesCell Differentiation processCellular ImmunityChronicChronic stressClinicalClinical ResearchCommunicable DiseasesCorticosteroneCorticotropinCyclic AMPDataDevelopmentDexamethasoneDiseaseEffector CellEndorphinsEnvironmentEquilibriumHPSE geneHarvestHelper-Inducer T-LymphocyteHumanHydrocortisoneImmuneImmune responseImmune systemImmunityImmunosuppressionImpairmentIn VitroIndividualInfectionInfusion proceduresInterleukin-2Interleukin-4Interleukin-5InvestigationKnockout MiceLife ExperienceLife StyleLinkMediatingMediator of activation proteinModelingMusNaloxoneNumbersOpioid PeptideOpioid ReceptorOpportunistic InfectionsOutcomePathologic ProcessesPhenotypePlayPneumococcal PneumoniaPopulationPredispositionProcessProductionProtein BiosynthesisPsychosocial FactorRegulationRisk FactorsRoleSpecificitySplenocyteStressSubstance abuse problemTherapeuticTherapeutic immunosuppressionThinkingTimeVaccinationVaccinesVirus DiseasesWild Type Mousebeta-Endorphinbiological adaptation to stresscytokinedesignemotional distressendogenous opioidshuman diseasehypothalamic-pituitary-adrenal axisimmune functionin vivo Modelmouse modelmu opioid receptorsneurochemistrypathogenprogramspsychologicreceptorresponserestraint stressrole modelstressortranscription factortumor

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