Project 3: Molecular Mechanisms and Signaling Pathways for T Cell Anergy

项目3：T细胞无能的分子机制和信号通路

• 批准号: 7500384
• 负责人: MICHAEL P CZECH
• 金额: $ 14.72万
• 依托单位: UNIV OF MASSACHUSETTS MED SCH WORCESTER
• 依托单位国家: 美国
• 财政年份: 2006
• 资助国家: 美国
• 起止时间: 2006-12-01 至 2007-11-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7500384
• 关键词: 1-Phosphatidylinositol 3-Kinase; Address; Animal Model; Animals; Antigens; Antisense RNA; Attenuated; Biological Models; CD8B1 gene; Candidate Disease Gene; Cell Line; Cell model; Cell physiology; Cells; Collaborations; Columbidae; Complement; Complex; Data; Databases; Elevation; Engraftment; Exhibits; Expressed Sequence Tags; Funding; Gene Expression; Genes; Genomics; Goals; Graft Tolerance; Guanine Nucleotide Exchange Factors; Helper-Inducer T-Lymphocyte; Hour; Human; Immune response; In Vitro; Interleukin-2; Knockout Mice; Laboratories; Lymphocyte; Lymphocyte Function; Maintenance; Mediating; Membrane; Methodology; Methods; Microarray Analysis; Microfilaments; Mitogen-Activated Protein Kinases; Molecular; Monoclonal Antibody HuM291; Muromonab-CD3; Mus; Normal Range; Peptides; Phosphoric Monoester Hydrolases; Play; Proliferating; Protein Kinase; Proteins; RNA; RNA Interference; Reagent; Reporting; Research Personnel; Role; Screening procedure; Signal Pathway; Signal Transduction; Signaling Protein; Skin; Skin graft; Specificity; T-Cell Activation; T-Lymphocyte; Techniques; Testing; Thinking; Time; Transfusion; Tumor Necrosis Factor Ligand Superfamily Member 6; Week; anergy; base; cell type; cytochrome c; day; design; gene discovery; in vivo; in vivo Model; islet; knockout gene; lymph nodes; novel; programs; rapid technique; research study; response; selective expression; skin allograft; transcription factor