Interleukin-18 and post infarct myocardial remodeling

Interleukin-18 与梗死后心肌重塑

• 批准号: 7784475
• 负责人: Chandrasekar Bysani
• 金额: --
• 依托单位: SOUTHEAST LOUISIANA VETERANS HEALTH CARE
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-04-01 至 2013-12-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7784475
• 关键词: 70-kDa Ribosomal Protein S6 Kinases; Activation Analysis; Acute; Acute myocardial infarction; Address; Angiotensin-Converting Enzyme Inhibitors; Apoptosis; Autoimmune Responses; Biochemical; Cardiac; Cardiac Myocytes; Cell Adhesion Molecules; Cell Death; Cells; Cessation of life; Chronic; Complex; Congestive Heart Failure; Data; Deposition; Deterioration; Development; Dilatation - action; Disease; Down-Regulation; Extracellular Matrix; Failure; Fibroblasts; Fibrosis; Genes; Goals; Growth Factor; Health; Hospitalization; Human; Hypertrophy; In Vitro; Incidence; Infarction; Inflammatory; Inflammatory Response; Injury; Interleukin-18; Interleukin-6; Knock-out; Knockout Mice; Left Ventricular Hypertrophy; Left Ventricular Remodeling; Measures; Mediating; Military Personnel; Molecular; Morbidity - disease rate; Mus; Myocardial; Myocardial Infarction; Myocardium; Outcome; PTEN gene; Pathologic Processes; Pathology; Patient Care; Patients; Performance; Phase; Phenotype; Play; Population; Process; Publishing; Reaction; Regulation; Reporting; Role; Signal Transduction; Therapeutic; Tissues; Transgenic Mice; Ventricular; Ventricular Remodeling; Veterans; abstracting; base; chemokine; cytokine; design; fetal; gain of function; human TSC2 protein; immune activation; improved; in vivo; inflammatory marker; injured; innovation; interleukin-18 receptor; loss of function; migration; mortality; myocardial infarct sizing; novel; overexpression; therapeutic target