课题基金 / 基金详情

NEURONAL AXONS (AXONS FROM DRG NEURONS)

NEURONAL AXONS (AXONS FROM DRG NEURONS)
神经元轴突（来自 DRG 神经元的轴突）
批准号：
8168600
负责人：
William C Mobley
金额：
$0.65万
依托单位：
BAYLOR COLLEGE OF MEDICINE
依托单位国家：
美国
项目类别：
财政年份：
2010
资助国家：
美国
项目状态：
已结题
起止时间：
2010-01-15 至 2010-12-31
来源：
https://reporter.nih.gov/project-details/8168600
关键词：
AxonCommunicationComputer Retrieval of Information on Scientific Projects DatabaseDataFundingGrantInstitutionNeuronsResearchResearch PersonnelResourcesSourceUnited States National Institutes of Health

项目摘要

项目成果

William C Mobley的其他基金

相似基金

相关文献

中文摘要
翻译
这个子项目是许多研究子项目中利用 资源由NIH/NCRR资助的中心拨款提供。子项目和 调查员(PI)可能从NIH的另一个来源获得了主要资金， 并因此可以在其他清晰的条目中表示。列出的机构是 该中心不一定是调查人员的机构。 轴突是货物运输和神经元之间交流的“高速公路”。了解神经退行性神经元的轴突与健康轴突的轴突亚结构变化是很重要的。这些轴突3D亚结构变化可以用冷冻采集的数据进行注释。
英文摘要
This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Axons are the "highways" of cargo transport and communication between neurons. It is important to understand the axonal sub-structural changes in neurodegenerating neurons' axons vs. healthy axons. These axonal 3D sub-structural changes can be annotated using cryo-ET acquired data.
期刊论文(0)
专著(0)
科研奖励(0)
会议论文
Antisense Oligonucleotides targeting APP to prevent neurodegeneration in models of Down Syndrome and Alzheimer's disease
Antisense Oligonucleotides targeting APP to prevent neurodegeneration in models of Down Syndrome and Alzheimer's disease
Antisense Oligonucleotides targeting APP to prevent neurodegeneration in models of Down Syndrome and Alzheimer's disease
Antisense Oligonucleotides targeting APP to prevent neurodegeneration in models of Down Syndrome and Alzheimer's disease
海外基金