Mechanisms of HTLV-1 p30 in Transcription and DMA Repair

HTLV-1 p30 转录和 DMA 修复机制

• 批准号: 8079527
• 负责人: MICHAEL D. LAIRMORE
• 金额: $ 19.16万
• 依托单位: OHIO STATE UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2003
• 资助国家: 美国
• 起止时间: 2003-04-21 至
• 项目状态: 未结题
• 来源: https://reporter.nih.gov/project-details/8079527
• 关键词: Acetylation; Adult; Amino Acid Motifs; Animals; Ataxia-Telangiectasia-Mutated protein kinase; Binding; Biological Assay; Biology; Cell Cycle; Cell Survival; Cell physiology; Cellular biology; Code; Complex; Consensus; DNA Repair; Data; Disease; EP300 gene; Environment; Equilibrium; Family member; Gagging; Genes; Genetic Transcription; Goals; Human; Human T-lymphotropic virus 1; Human T-lymphotropic virus 2; Infection; Lymphocyte; Lymphocyte Activation; Maintenance; Malignant Neoplasms; Mediating; Messenger RNA; Modeling; Modification; Molecular Cloning; Mutation; Nuclear; Nucleolar Proteins; Open Reading Frames; Oryctolagus cuniculus; Phase; Post-Transcriptional Regulation; Process; Proteins; Publishing; Retroviridae; Role; Signal Transduction; Site; T-Cell Leukemia; T-Cell Lymphoma; T-Lymphocyte; Taxes; Testing; Therapeutic Intervention; Transactivation; Transcriptional Regulation; Translations; Viral; Viral Proteins; Virus; adult leukemia; base; cell transformation; comparative; computer studies; env Gene Products; in vivo; insight; leukemia; novel; ranpirnase; repaired; tool; transcription factor; transmission process