New Signaling pathways that positively and negatively regulate CD4 T cells via th

通过 th 正向和负向调节 CD4 T 细胞的新信号通路

• 批准号: 8742789
• 负责人: EDWARD Y SKOLNIK
• 金额: $ 15.48万
• 依托单位: NEW YORK UNIVERSITY SCHOOL OF MEDICINE
• 依托单位国家: 美国
• 财政年份: 2013
• 资助国家: 美国
• 起止时间: 2013-12-30 至 2015-12-29
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8742789
• 关键词: 1-Phosphatidylinositol 3-Kinase; Antigen-Presenting Cells; Antigens; Autoimmune Diseases; B-Cell Activation; Binding; Biological; CD4 Positive T Lymphocytes; Calcium-Activated Potassium Channel; Cellular biology; Data; Development; Diphosphates; Disease; Drug Targeting; Feedback; Goals; Graft Rejection; Histidine; Human; In Vitro; Learning; Lipid Bilayers; Location; Mediating; Nucleoside diphosphate kinase B; Pathway interactions; Phosphoric Monoester Hydrolases; Phosphotransferases; Play; Protein Isoforms; Proteins; Receptor Activation; Recruitment Activity; Regulation; Role; Signal Pathway; T-Cell Activation; T-Cell Receptor; T-Lymphocyte; Viral Tumor Antigens; abstracting; immunological synapse; in vivo; inhibitor/antagonist; myotubularin; novel; overexpression; phosphatidylinositol 3-phosphate; phosphohistidine; protein-histidine kinase; release of sequestered calcium ion into cytoplasm

No abstract provided

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