Molecular mechanisms of low-level vancomycin resistance in clinical Staphylococcus aureus

临床金黄色葡萄球菌低水平万古霉素耐药的分子机制

• 批准号: nhmrc : 628652
• 负责人: Prof Benjamin Howden
• 金额: $ 29.2万
• 依托单位: The University of Melbourne
• 依托单位国家: 澳大利亚
• 项目类别: NHMRC Project Grants
• 财政年份: 2010
• 资助国家: 澳大利亚
• 起止时间: 2010-01-01 至 2012-12-31
• 项目状态: 已结题
• 来源: https://researchdata.edu.au/molecular-mechanisms-low-staphylococcus-aureus/100526
• 关键词: Molecular; mechanisms; low; level; vancomycin

The common bacteria Staphylococcus aureus causes many infections in humans, and is becoming more resistant to antibiotic treatments, especially in hospitals. This project will determine how this bacteria is developing resistance to some of our last available antibiotics. This will provide an important basis for detecting and preventing this antibiotic resistance problem in future.

常见细菌金黄色葡萄球菌会引起人类多种感染，并且对抗生素治疗的耐药性越来越强，尤其是在医院。该项目将确定这种细菌如何对我们最后可用的一些抗生素产生耐药性。这将为今后检测和预防抗生素耐药性问题提供重要依据。