Protective and Deleterious Roles of Regulated Exocytosis from Mast Cells

肥大细胞胞吐作用的保护作用和有害作用

• 批准号: 8122832
• 负责人: Roberto Adachi
• 金额: $ 36.38万
• 依托单位: UNIVERSITY OF TX MD ANDERSON CAN CTR
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-08-20 至 2011-07-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8122832
• 关键词: Allergic; Allergic inflammation; Animal Model; Asthma; Bacteria; Bacterial Infections; Biochemical; Biological Assay; Cell Degranulation; Cells; Cellular biology; Chronic; Data; Defect; Development; Disease; Eicosanoids; Evolution; Exocytosis; Extrinsic asthma; Failure; Functional disorder; Genetic Polymorphism; Helminths; Human Genetics; Hypersensitivity; Immune; Immune response; Impairment; Individual; Infection; Infectious Agent; Inflammation; Inflammation Mediators; Inflammatory; Left; Modeling; Modification; Morbidity - disease rate; Morphology; Mus; Mutant Strains Mice; Mutation; Peritonitis; Play; Pneumonia; Process; Property; Protein Isoforms; Proteins; Reagent; Relative (related person); Rheumatoid Arthritis; Role; Secretory Vesicles; Structure; Testing; Therapeutic; Toxic effect; Transgenic Animals; Transgenic Mice; Transgenic Organisms; Tryptase; anti-IgE; cytokine; gain of function; gain of function mutation; genetic manipulation; lipid mediator; mast cell; mutant; novel diagnostics; protective effect; public health relevance; research study; response; syntaxin binding protein 1; therapeutic target