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Pathogenic Role of Islet Cell Autoantibodies in Type I Diabetes

Pathogenic Role of Islet Cell Autoantibodies in Type I Diabetes
胰岛细胞自身抗体在 I 型糖尿病中的致病作用
批准号：
8278713
负责人：
Raphael A. Clynes
金额：
$12.0万
依托单位：
COLUMBIA UNIVERSITY HEALTH SCIENCES
依托单位国家：
美国
项目类别：
财政年份：
2005
资助国家：
美国
项目状态：
已结题
起止时间：
2005-04-01 至 2012-06-30
来源：
https://reporter.nih.gov/project-details/8278713
关键词：
AddressAnimal ModelAntibodiesAntigen PresentationAntigen TargetingAntigen-Presenting CellsAntigensArthritisAutoantibodiesAutoantigensAutoimmune ProcessAutoimmune ResponsesAutoimmunityB-Cell DevelopmentB-LymphocytesCellsClinicClinicalCross-PrimingDataDendritic Cell PathwayDendritic CellsDevelopmentDiabetes MellitusDiseaseDisease ProgressionExperimental Autoimmune EncephalomyelitisFc ReceptorFundingGeneticHumanITGAX geneImmunosuppressive AgentsInbred NOD MiceInflammationInflammatoryInflammatory ResponseInjuryInsulin-Dependent Diabetes MellitusInterferonsInterleukin-10Islet CellLymphocyteMediatingMediator of activation proteinModelingMusMyeloid CellsNational Institute of Diabetes and Digestive and Kidney DiseasesNephritisOralPathogenesisPathway interactionsPhosphotransferasesPopulationProcessProtein Tyrosine KinaseReceptor SignalingReceptors, Antigen, B-CellRoleSYK geneSignal PathwaySignal TransductionSpleenSplenocyteSystemic Lupus ErythematosusT-LymphocyteTimeTissuesTyrosine Kinase InhibitorUnited States National Institutes of HealthWorkabstractingautoreactive T cellcellular targetingclinically relevanthuman FCGR2A proteinhuman SYK proteinin vivoinhibitor/antagonistinsightmeetingsreceptorsmall moleculesuccesstherapeutic target

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