Mechanisms of Cellular Immune Dysfunction in Patients with Hepatitis C Virus and

丙型肝炎病毒和病毒感染患者细胞免疫功能障碍的机制

• 批准号: 7908872
• 负责人: Kyong-Mi Chang
• 金额: --
• 依托单位: PHILADELPHIA VA MEDICAL CENTER
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-10-01 至 2014-03-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7908872
• 关键词: Acute Hepatitis C; Blood; CD4 Positive T Lymphocytes; CTLA4 gene; Cessation of life; Chronic; Chronic Hepatitis C; Cirrhosis; Clinical; Cytotoxic T-Lymphocyte-Associated Protein 4; Data; Dendritic Cells; Development; Disease Progression; Evolution; Frequencies; Functional disorder; General Population; HIV; Health; Healthcare; Hepatitis C; Hepatitis C virus; Immune; Immune System Diseases; Immunotherapeutic agent; Infection; Interleukin-10; Ligands; Literature; Liver Cirrhosis; Liver diseases; Malignant neoplasm of liver; Monitor; Morbidity - disease rate; Outcome; Pathway interactions; Patients; Peripheral; RNA Viruses; Regulation; Regulatory Pathway; Regulatory T-Lymphocyte; Relative (related person); Role; Sampling; Seroprevalences; Signal Transduction; T cell response; T-Lymphocyte; Therapeutic; Transforming Growth Factor beta; Veterans; Virus; Virus Diseases; base; cytokine; insight; mortality; programs; public health relevance; receptor; receptor expression; response; virus pathogenesis