Molecular mechanism of the human copper-transporting ATPases

人类铜转运ATP酶的分子机制

• 批准号: 177538
• 负责人: Dmitriev Oleg Y
• 金额: $ 9.63万
• 依托单位: University of Saskatchewan
• 依托单位国家: 加拿大
• 项目类别: Operating Grants
• 财政年份: 2008
• 资助国家: 加拿大
• 起止时间: 2008-09-01 至 2010-09-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=177538&lang=en
• 关键词: Atpase; Copper Transport; Menkes Disease; Nmr; Protein Stucture; Wilson Disease

Copper is required for the activity of many enzymes involved in respiration, neuron function, formation of connective tissue, endocrine processes, and radical detoxification in the human body. Wilson disease ATPase and Menkes disease ATPase regulate copp

铜是人体呼吸、神经元功能、结缔组织形成、内分泌过程和自由基解毒等许多酶的活性所必需的。 肝豆状核变性ATP酶和门克斯病ATP酶调节copp