Matrix metalloproteinase-2 (MMP-2) is activated during ischemia in a model of myocardial infarction

心肌梗死模型缺血期间基质金属蛋白酶 2 (MMP-2) 被激活

• 批准号: 253375
• 负责人: De Jesus Cadete Virgilio J
• 金额: $ 0.09万
• 依托单位: University of Saskatchewan
• 依托单位国家: 加拿大
• 财政年份: 2012
• 资助国家: 加拿大
• 起止时间: 2012-01-01 至 2012-02-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=253375&lang=en
• 关键词: Cardioprotection; Contractile Dysfunction; Synergy

During a heart attack (ischemia) the proteins responsible for heart beating (contractile proteins) are destroyed and this leads to irregular beating (contractile dysfunction). We have found that degradation of contractile proteins is performed by a degrad

在心脏病发作（缺血）期间，负责心脏跳动的蛋白质（收缩蛋白）被破坏，这导致不规则的跳动（收缩功能障碍）。我们发现，收缩蛋白的降解是由一种降解蛋白完成的。