Evading and subverting host cell machineries to ensure HIV-1 genomic RNA fate

逃避和破坏宿主细胞机制以确保 HIV-1 基因组 RNA 的命运

• 批准号: 254794
• 负责人: Mouland Andrew J
• 金额: $ 57.66万
• 依托单位: Lady Davis Institute for Medical Research (Mtl)
• 依托单位国家: 加拿大
• 项目类别: Operating Grants
• 财政年份: 2012
• 资助国家: 加拿大
• 起止时间: 2012-03-01 至 2017-03-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=254794&lang=en
• 关键词: Endosomes; Hiv-1/Aids; Intracellular Trafficking; Nuclear Pore Proteins; Nup62; Proteomics And Mass Spectrometry; Rilp; Upf Proteins; Viral Evasion Strategies; Virus-Host Interactions

HIV-1 is the causative agent that causes acquired immunodeficiency syndrome or AIDS. Since 1981, more than 25 million people have died of AIDS. It is estimated that close to 44 million people worldwide are infected (United Nations Programme on HIV/AIDS) a

HIV-1是导致获得性免疫缺陷综合症或艾滋病的病原体。自1981年以来，已有2500多万人死于艾滋病。据估计，全世界有近4400万人受到感染(联合国艾滋病毒/艾滋病规划署)a